Treatment of these lesions would be required to achieve prophylaxis against the development of RD. There are peripheral retinal lesions such as retinal breaks or lattice degeneration that can predispose to RD. Their subsequent management plays an important role in reducing the risk of RD, especially under special circumstances such as prior to refractive surgery or any intraocular procedure or in patients with abnormal vitreous such as in Sticker syndrome or Marfan syndrome. Thus, it is important to properly evaluate the retinal periphery for the presence of PRDs. This can lead to the development of retinal tears and cause RD. Anomalous, acute, symptomatic PVD with recent-onset flashes and floaters (within 3 months duration), in the presence or absence of PRD, can lead to various deleterious effects on the retina as a result of abnormal traction at the vitreoretinal interface. The development of PVD usually occurs either spontaneously in old age, as seen in most cases, or can be brought about by events such as cataract and refractive surgeries, intravitreal injections, retinal trauma, uveitis, pan-retinal photocoagulation, laser capsulotomy and syndromic diseases such as Marfan syndrome or Stickler syndrome. PVD is defined as the separation of the posterior cortical vitreous from the internal limiting membrane of the retina and is the most common cause of floaters. Posterior vitreous detachment (PVD) plays a crucial role in precipitating RD in eyes with PRDs. can result in rhegmatogenous retinal detachment (RD). While most of them are clinically insignificant, a few of these degenerations such as lattice degeneration, degenerative retinoschisis, peripheral retinal tears, cystic retinal tufts and, rarely, zonular traction tufts. Peripheral retinal degeneration (PRD) is a broad term which includes various lesions such as the lattice degeneration, snail-track degeneration, snowflake degeneration, atrophic or operculated retinal holes, peripheral retinal tears, senile retinoschisis, white and dark without pressure areas, paving stone degeneration and peripheral cystoid degeneration.
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